Kumar wrote:
Andrew B. Chung, MD/PhD wrote:
Kumar wrote:
Andrew B. Chung, MD/PhD wrote:
Kumar wrote:
Andrew B. Chung, MD/PhD wrote:
Kumar wrote:
Andrew B. Chung, MD/PhD wrote:
Kumar wrote:
snip> > > > > > Whether new arteries by angiogenesis always occur on
narrowing and
obstructtions of Cor. arteries?
New collateral arteries do not form when they are not needed.
When needed, say in case of narrowing or obstrucion of blood flow?
Correct.
How EECP can help in forming new collateral arteries or increase in
size? On the contrary it may discourage it?
It is possible that EECP may be helping to mobilize stem cells to form
these new vessels.
How?
There are stem cells residing in the arteries of the legs.
When blood supply is improved, why trigger to new vessels will be
needed?
The improvement in myocardial perfusion is transient and achieved by
raising pressures during diastole. The obstructions remain present
during EECP and so the need for collateral vessels will be apparent to
the possible relocalization of stem cells from the leg arteries to the
coronaries.
Will it also not happen without EECP?
Whether manipulating increase in blood flow by invasive or non-invasive
means discourage forming of collateral arteries and also decrease in
their size?
Collateral arteries are the non-invasive means of increasing blood
flow.
Yes, but Collateral arteries are formed due to decrease in blood flow?
They are formed where there is need, LORD willing.
What is stumuli to them?
The presence of the obstructive atheroclerotic coronary lesions
I think new Collateral arteries can be from upstream to downstream?
There is slowing of blood flow upstream of the obstructive lesion.
Can we also consider it as "decreased supply of nutrients/O2" ?
That would be downstream of where the collaterals will need to start
forming.
Will "decreased supply of nutrients/O2" be not due to upstream or/and
downstream reasons?
This will be downstream.
If there is a slowing of blood flow upstream of the obstructive
lesion, what can be the use of collaterals from downstream?
The collaterals will be coming from upstream to supply the deficit
downstream.
Some fats( saturated) are directly travell in circulation whereas other
via long process through lympatic system? Do such differences impact
our health in any way?
Not clinically seen.
Is it true that some saturated fats travel in circulation after
absorption directly whereas most other via lymph system route?
Clinical importance not established.
Can we consider such effects as effect of free fatty acids?
Lack of clinical importance is not an effect.
In what form, abosrbed fatty acids part in circulating or stored
triglycerides exists in body? Its chain and bond still remain same as
absorbed or processed and changed to different forms prior to their
oxidation and use for energy?
As lipoprotein particles.
Lipid profile also interpret triglygirides? Which type of chain is
there in fatty acid part of triglyceride present in blood or stored?
VLDL.
Can cholesterols in body be synthesized from fatty acids OR if they
need to be directly absorbed in intestines??
Cholesterols can be synthesized de novo from fats, carbohydrates, and
protein.
snip> > > > To compare, whch is more harmful out of saturated and
unsaturated fats
for either their storing capacity or free radicle/oxidative stress
causing capacity?
It is wiser to focus on ridding the VAT.
Just for understanding purpose.
For benefit.
Yes, but I meant which out of Saturated(esp. milk fat) and unsaturated
is more beneficial to diabetic2 without abnormal lipid profile? Few
thought that milkfat causes more bile secretion in intestine and more
of its excretion, so may cause usage and recirculation of cholesterols
and their control? Is it right?
It remains wise to focus on ridding the VAT.
Is it wise to take oils/foods with no cholesterol?
Not as wise as eating less.
snip> > Bit inconvenient/difficult but Can we eat more but still
optimal by
manipulating specific foods?
Not difficult for those who are using the 2PD-OMER Approach and have
overcome the brainwashing that "hunger is bad."
Can decreased blood flow to parts as a result to vasoconstriction or
decrease in size and numbers of arteries sypplying to tissues trigger
overeating?
Not clinically seen.
snip> > > > > > What are bile abnormalities other than decreased flow
from liver or
gall bladder?
Still wiser to let your doctor(s) figure this out for you.
I don't have such abnormilty but want to understand, one reson why
ancient systems gave so much importance to bile?
This is what they could see with their eyes when they disembowelled
people.
That is measuring/understanding the imbalance. But what can be the
imbalances and disorders related to these?
None clinically seen.
How more or less secretion of bile and more or less of its
concentration can impact health?
The secretion of bile is not involved in the development and
persistence of type-2 diabetes.
It can effect fats absorption, more fats may be a reason to getting IR
?
More VAT from overeating is the reason for the IR.
What is the contribution of absorbed fats in forming VAT?
When there is no overeating, there is no VAT being formed.
Whether amount, composition
and reabsorption of bile changes on
different exposures & abnormilities?
Still wiser to let your doctor(s) figure this out for you.
As above?
Yes.
Whether low gastric acid or excess bile is the reason to absorbing more
fats & IBS?
Not clinically seen.
What physiological effects, variations in atmospheric temperature &
moisture/humidity to which we are exposed can cause? Can high temp.
cause somewhat vasodilating and high humidity incread BP or
vasoconstricting effects, low temp. and low humidity opposite?
These effects are not clinically meaningful.
Can taking sauna or steam bath attract water from body benefitting to
those with cells swellings, edema etc?
Not clinically seen.
Can these effect oedma, cell's/intimal swellings?
Not clinically seen.
How sauna and steam bath differ on health impact?
Not clinically relevant.
Whether Prespitation from these,can benefit by removing water from
cells if abnormal due to hypotonic environment out of different cells?
Or, can such prespiration reverse hypotonicy?
Still not clinically relevant.
Can excessive prespiration express vasodilation whereas excess urine
vasoconstriction?
Not clinically seen.
May GOD conitnue to heal your heart by curing your diabetes, dear
neighbor Kumar whom I love unconditionally.
Prayerfully in Christ's amazing love,
Andrew <><
--
Andrew B. Chung
Cardiologist, Atlanta, Georgia, USA
http://HeartMDPhD.com/HolySpirit
As for knowing who are the very elect, these you will know by the
unconditional love they have for everyone including their enemies
(Matthew 5:44-45, 1 Corinthians 13:3, James 2:14-17).
http://HeartMDPhD.com/Love
.
